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ARTICLE TYPE : REVIEW ARTICLE

Published on :   10 Oct 2026, Volume - 2
Journal Title :   WebLog Journal of Thyroid Research | WebLog J Thyroid Res | WJTR
Source URL:   weblog icon https://weblogoa.com/articles/wjtr.2026.j1002
Permanent Identifier (DOI) :   doi icon https://doi.org/10.5281/zenodo.23336387

State of Art Antithyroid Drug-Induced Neutropenia and Agranulocytosis

Emmanuel Andrès 1,2,3 *
Xavier Jannot 1,2
Jean-Edouard Terrade 1,2
Khalid Serraj 4
Noel Lorenzo-Villalba 1,2
1Department of Internal Medicine, Hôpital de Hautepierre, University Hospital of Strasbourg, 67084 Strasbourg, France
2Referral Centre of Immune Cytopenias, University Hospital of Strasbourg, 67084 Strasbourg, France
3Institute of Physiology, FMTS-EA 3072, Faculty of Medicine, University of Strasbourg, 67000 Strasbourg, France
4Department of Internal Medicine, University Hospital of Oujda, 59000 Oujda, Morocco

Abstract

Antithyroid Drugs (ATDs) - principally carbimazole, methimazole (thiamazole), and propylthiouracil - remain central to the treatment of hyperthyroidism but can rarely cause idiosyncratic neutropenia and agranulocytosis, a potentially fatal adverse drug reaction. Agranulocytosis is conventionally defined by an absolute neutrophil count below 0.5×109/L and may develop abruptly after a previously normal blood count. This Seminar reviews contemporary epidemiology, drug- and patient-related risk factors, mechanisms, pharmacogenomic susceptibility, clinical presentation, differential diagnosis, monitoring, and management. Risk is heterogeneous and reflects ATD exposure, dose, age, population, and genetic background. HLA-B*38:02 and HLA-DRB1*08:03 are strongly associated with susceptibility in several Asian populations, whereas HLA-B*27:05 is particularly relevant in European cohorts; these associations are not yet sufficient to support universal pre-treatment screening. Fever, sore throat, or other infectious symptoms during ATD treatment should prompt immediate blood-cell assessment, and confirmed or strongly suspected agranulocytosis requires immediate withdrawal of the culprit drug. Febrile profound neutropenia is a medical emergency requiring prompt empirical antimicrobial therapy. G-CSF can accelerate neutrophil recovery in selected patients, although an independent mortality benefit remains unproven. After true ATD-induced agranulocytosis, re-exposure should be avoided and definitive treatment with radioiodine or thyroidectomy should be considered. Prevention therefore rests primarily on patient education and rapid symptom-triggered testing, with selective periodic monitoring where local circumstances justify it. Prospective validation of population-specific pharmacogenomic and clinical risk models is now needed to determine whether earlier detection or genotype-guided treatment improves patient-important outcomes.

Keywords: Antithyroid Drugs; Carbimazole; Methimazole; Propylthiouracil; Neutropenia; Agranulocytosis; Graves' Disease; Pharmacogenomics; G-CSF; Pharmacovigilance

Citation

Andrès E, Jannot X, Terrade J-E, Serraj K, Lorenzo-Villalba N. State of Art Antithyroid Drug-Induced Neutropenia and Agranulocytosis. WebLog J Thyroid Res. wjtr.2026.j1002. https://doi.org/10.5281/zenodo.23336387